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ArticlesPeer ReviewedFull Text
Genetic Risk in Chronic Pancreatitis: The Trypsin-Dependent Pathway
Author / Creator:Hegyi, Eszter ; Sahin-Tóth, Miklós
Publisher:New York: Springer US
Journal title:Digestive diseases and sciences, 2017-07, Vol.62 (7), p.1692-1701
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Human CPA1 mutation causes digestive enzyme misfolding and chronic pancreatitis in mice
Author / Creator:Hegyi, Eszter ; Sahin-Tóth, Miklós
Publisher:England: BMJ Publishing Group LTD
Journal title:Gut, 2019-02, Vol.68 (2), p.301-312
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A preclinical model of chronic pancreatitis driven by trypsinogen autoactivation
Author / Creator:Geisz, Andrea ; Sahin-Tóth, Miklós
Publisher:London: Nature Publishing Group UK
Journal title:Nature communications, 2018-11, Vol.9 (1), p.5033-9, Article 5033
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Misfolding-induced chronic pancreatitis in CPA1 N256K mutant mice is unaffected by global deletion of Ddit3/Chop
Author / Creator:Németh, Balázs Csaba ; Demcsák, Alexandra ; Geisz, Andrea ; Sahin-Tóth, Miklós
Publisher:London: Nature Publishing Group UK
Journal title:Scientific reports, 2022-04, Vol.12 (1), p.6357-6357, Article 6357
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Bicarbonate defective CFTR variants increase risk for chronic pancreatitis: A meta-analysis
Author / Creator:Berke, Gergő ; Gede, Noémi ; Szadai, Letícia ; Ocskay, Klementina ; Hegyi, Péter ; Sahin-Tóth, Miklós ; Hegyi, Eszter
Publisher:United States: Public Library of Science
Journal title:PloS one, 2022-10, Vol.17 (10), p.e0276397
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Prospective, Multicentre, Nationwide Clinical Data from 600 Cases of Acute Pancreatitis
Author / Creator:Párniczky, Andrea ; Kui, Balázs ; Szentesi, Andrea ; Balázs, Anita ; Szűcs, Ákos ; Mosztbacher, Dóra ; Czimmer, József ; Sarlós, Patrícia ; Bajor, Judit ; Gódi, Szilárd ; Vincze, Áron ; Illés, Anita ; Szabó, Imre ; Pár, Gabriella ; Takács, Tamás ; Czakó, László ; Szepes, Zoltán ; Rakonczay, Zoltán ; Izbéki, Ferenc ; Gervain, Judit ; Halász, Adrienn ; Novák, János ; Crai, Stefan ; Hritz, István ; Góg, Csaba ; Sümegi, János ; Golovics, Petra ; Varga, Márta ; Bod, Barnabás ; Hamvas, József ; Varga-Müller, Mónika ; Papp, Zsuzsanna ; Sahin-Tóth, Miklós ; Hegyi, Péter
Publisher:United States: Public Library of Science
Journal title:PloS one, 2016-10, Vol.11 (10), p.e0165309-e0165309
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Loss of chymotrypsin-like protease (CTRL) alters intrapancreatic protease activation but not pancreatitis severity in mice
Author / Creator:Mosztbacher, Dóra ; Jancsó, Zsanett ; Sahin-Tóth, Miklós
Publisher:London: Nature Publishing Group UK
Journal title:Scientific reports, 2020-07, Vol.10 (1), p.11731-11731, Article 11731
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Chymotrypsin C (caldecrin) promotes degradation of human cationic trypsin: Identity with Rinderknecht's enzyme Y
Author / Creator:Szmola, Richárd ; Sahin-Tóth, Miklós
Publisher:United States: National Academy of Sciences
Journal title:Proceedings of the National Academy of Sciences - PNAS, 2007-07, Vol.104 (27), p.11227-11232
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Engineering mouse cationic trypsinogen for rapid and selective activation by cathepsin B
Author / Creator:Demcsák, Alexandra ; Geisz, Andrea ; Sahin-Tóth, Miklós
Publisher:London: Nature Publishing Group UK
Journal title:Scientific reports, 2019-06, Vol.9 (1), p.9188-12, Article 9188
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Risk of chronic pancreatitis in carriers of loss-of-function CTRC variants: A meta-analysis
Author / Creator:Takáts, Amanda ; Berke, Gergő ; Gede, Noémi ; Németh, Balázs Csaba ; Witt, Heiko ; Głuszek, Stanisław ; Rygiel, Agnieszka Magdalena ; Hegyi, Péter ; Sahin-Tóth, Miklós ; Hegyi, Eszter
Publisher:United States: Public Library of Science
Journal title:PloS one, 2022-05, Vol.17 (5), p.e0268859-e0268859
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Carboxyl ester lipase hybrid 1 (CEL-HYB1) haplotypes confer varying risk for chronic pancreatitis
Author / Creator:Berke, Gergő ; Sándor, Máté ; Xiao, Xunjun K. ; Lowe, Mark E. ; Ewers, Maren ; Erőss, Bálint ; Masson, Emmanuelle ; Németh, Balázs Csaba ; Vincze, Áron ; Czakó, László ; Rygiel, Agnieszka Magdalena ; Rosendahl, Jonas ; Chen, Jian-Min ; Witt, Heiko ; Hegyi, Péter ; Sahin-Tóth, Miklós ; Hegyi, Eszter
Publisher:London: Nature Publishing Group UK
Journal title:Scientific reports, 2024-12, Vol.14 (1), p.30965-7, Article 30965
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Pancreatitis-associated chymotrypsinogen C (CTRC) mutant elicits endoplasmic reticulum stress in pancreatic acinar cells
Author / Creator:Szmola, Richárd ; Sahin-Tóth, Miklós
Publisher:London: BMJ Publishing Group Ltd and British Society of Gastroenterology
Journal title:Gut, 2010-03, Vol.59 (3), p.365-372
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CELA2A mutations predispose to early-onset atherosclerosis and metabolic syndrome and affect plasma insulin and platelet activation
Author / Creator:Esteghamat, Fatemehsadat ; Broughton, James S. ; Smith, Emily ; Cardone, Rebecca ; Tyagi, Tarun ; Guerra, Mateus ; Szabó, András ; Ugwu, Nelson ; Mani, Mitra V. ; Azari, Bani ; Kayingo, Gerald ; Chung, Sunny ; Fathzadeh, Mohsen ; Weiss, Ephraim ; Bender, Jeffrey ; Mane, Shrikant ; Lifton, Richard P. ; Adeniran, Adebowale ; Nathanson, Michael H. ; Gorelick, Fred S. ; Hwa, John ; Sahin-Tóth, Miklós ; Belfort-DeAguiar, Renata ; Kibbey, Richard G. ; Mani, Arya
Publisher:New York: Nature Publishing Group US
Journal title:Nature genetics, 2019-08, Vol.51 (8), p.1233-1243
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ArticlesPeer ReviewedFull Text
The common truncation variant in pancreatic lipase related protein 2 (PNLIPRP2) is expressed poorly and does not alter risk for chronic pancreatitis
Author / Creator:Németh, Balázs Csaba ; Pesei, Zsófia Gabriella ; Hegyi, Eszter ; Szücs, Ákos ; Szentesi, Andrea ; Hegyi, Péter ; Lowe, Mark E ; Sahin-Tóth, Miklós
Publisher:United States: Public Library of Science
Journal title:PloS one, 2018-11, Vol.13 (11), p.e0206869
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ArticlesPeer ReviewedFull Text
Autoantibodies against the exocrine pancreas in autoimmune pancreatitis: gene and protein expression profiling and immunoassays identify pancreatic enzymes as a major target of the inflammatory proces...
Author / Creator:Löhr, J-Matthias ; Faissner, Ralf ; Koczan, Dirk ; Bewerunge, Peter ; Bassi, Claudio ; Brors, Benedikt ; Eils, Roland ; Frulloni, Luca ; Funk, Anette ; Halangk, Walter ; Jesenofsky, Ralf ; Jesnowski, Ralf ; Kaderali, Lars ; Kleeff, Jörg ; Krüger, Burkhard ; Lerch, Markus M ; Lösel, Ralf ; Magnani, Mauro ; Neumaier, Michael ; Nittka, Stephanie ; Sahin-Tóth, Miklós ; Sänger, Julian ; Serafini, Sonja ; Schnölzer, Martina ; Thierse, Hermann-Josef ; Wandschneider, Silke ; Zamboni, Giuseppe ; Klöppel, Günter
Publisher:United States: Wolters Kluwer Health Medical Research, Lippincott Williams & Wilkins
Journal title:The American journal of gastroenterology, 2010-09, Vol.105 (9), p.2060-2071
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Mutations of human cationic trypsinogen (PRSS1) and chronic pancreatitis
Author / Creator:Teich, Niels ; Rosendahl, Jonas ; Tóth, Miklós ; Mössner, Joachim ; Sahin-Tóth, Miklós
Publisher:Hoboken: Wiley Subscription Services, Inc., A Wiley Company
Journal title:Human mutation, 2006-08, Vol.27 (8), p.721-730
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Chronic pancreatitis: Multicentre prospective data collection and analysis by the Hungarian Pancreatic Study Group
Author / Creator:Szücs, Ákos ; Marjai, Tamás ; Szentesi, Andrea ; Farkas, Nelli ; Párniczky, Andrea ; Nagy, György ; Kui, Balázs ; Takács, Tamás ; Czakó, László ; Szepes, Zoltán ; Németh, Balázs Csaba ; Vincze, Áron ; Pár, Gabriella ; Szabó, Imre ; Sarlós, Patrícia ; Illés, Anita ; Gódi, Szilárd ; Izbéki, Ferenc ; Gervain, Judit ; Halász, Adrienn ; Farkas, Gyula ; Leindler, László ; Kelemen, Dezső ; Papp, Róbert ; Szmola, Richárd ; Varga, Márta ; Hamvas, József ; Novák, János ; Bod, Barnabás ; Sahin-Tóth, Miklós ; Hegyi, Péter
Publisher:United States: Public Library of Science
Journal title:PloS one, 2017-02, Vol.12 (2), p.e0171420-e0171420
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Intron-mediated enhancement of SPINK1 expression for pancreatitis therapy
Author / Creator:Berke, Gergő ; Sahin-Tóth, Miklós
Publisher:London: BMJ Publishing Group LTD
Journal title:Gut, 2025-01, p.gutjnl-2024-332818
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ArticlesPeer ReviewedFull Text
Missense mutations in pancreatic secretory trypsin inhibitor (SPINK1) cause intracellular retention and degradation
Author / Creator:Király, Orsolya ; Wartmann, Thomas ; Sahin-Tóth, Miklós
Publisher:London: BMJ Publishing Group Ltd and British Society of Gastroenterology
Journal title:Gut, 2007-10, Vol.56 (10), p.1433-1438
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ArticlesPeer ReviewedFull Text
Uncertainties in the classification of human cationic trypsinogen (PRSS1) variants as hereditary pancreatitis-associated mutations
Author / Creator:Szmola, Richárd ; Sahin-Tóth, Miklós
Publisher:London: BMJ Publishing Group Ltd
Journal title:Journal of medical genetics, 2010-05, Vol.47 (5), p.348-350
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Showing 1 to 20 of 67 results

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Genetic Risk in Chronic Pancreatitis: The Trypsin-Dependent Pathway

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Human CPA1 mutation causes digestive enzyme misfolding and chronic pancreatitis in mice

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A preclinical model of chronic pancreatitis driven by trypsinogen autoactivation

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Misfolding-induced chronic pancreatitis in CPA1 N256K mutant mice is unaffected by global deletion of Ddit3/Chop

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Bicarbonate defective CFTR variants increase risk for chronic pancreatitis: A meta-analysis

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Prospective, Multicentre, Nationwide Clinical Data from 600 Cases of Acute Pancreatitis

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Loss of chymotrypsin-like protease (CTRL) alters intrapancreatic protease activation but not pancreatitis severity in mice

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Chymotrypsin C (caldecrin) promotes degradation of human cationic trypsin: Identity with Rinderknecht's enzyme Y

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Engineering mouse cationic trypsinogen for rapid and selective activation by cathepsin B

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Risk of chronic pancreatitis in carriers of loss-of-function CTRC variants: A meta-analysis

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Carboxyl ester lipase hybrid 1 (CEL-HYB1) haplotypes confer varying risk for chronic pancreatitis

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Pancreatitis-associated chymotrypsinogen C (CTRC) mutant elicits endoplasmic reticulum stress in pancreatic acinar cells

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CELA2A mutations predispose to early-onset atherosclerosis and metabolic syndrome and affect plasma insulin and platelet activation

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The common truncation variant in pancreatic lipase related protein 2 (PNLIPRP2) is expressed poorly and does not alter risk for chronic pancreatitis

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Autoantibodies against the exocrine pancreas in autoimmune pancreatitis: gene and protein expression profiling and immunoassays identify pancreatic enzymes as a major target of the inflammatory proces...

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Mutations of human cationic trypsinogen (PRSS1) and chronic pancreatitis

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Chronic pancreatitis: Multicentre prospective data collection and analysis by the Hungarian Pancreatic Study Group

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Intron-mediated enhancement of SPINK1 expression for pancreatitis therapy

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Missense mutations in pancreatic secretory trypsin inhibitor (SPINK1) cause intracellular retention and degradation

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Uncertainties in the classification of human cationic trypsinogen (PRSS1) variants as hereditary pancreatitis-associated mutations

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