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ArticlesPeer ReviewedFull Text
Latent Epstein-Barr virus infection collaborates with Myc over-expression in normal human B cells to induce Burkitt-like Lymphomas in mice
Author / Creator:Bristol, Jillian A. ; Nelson, Scott E. ; Ohashi, Makoto ; Casco, Alejandro ; Hayes, Mitchell ; Ranheim, Erik A. ; Pawelski, Abigail S. ; Singh, Deo R. ; Hodson, Daniel J. ; Johannsen, Eric C. ; Kenney, Shannon C.
Publisher:United States: Public Library of Science
Journal title:PLoS pathogens, 2024-04, Vol.20 (4), p.e1012132-e1012132
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The EphA2 receptor is activated through induction of distinct, ligand-dependent oligomeric structures
Author / Creator:Singh, Deo R. ; Kanvinde, Pranjali ; King, Christopher ; Pasquale, Elena B. ; Hristova, Kalina
Publisher:London: Nature Publishing Group UK
Journal title:Communications biology, 2018-02, Vol.1 (1), p.15-15, Article 15
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Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes
Author / Creator:Singh, Deo R. ; Nelson, Scott E. ; Pawelski, Abigail S. ; Cantres-Velez, Juan A. ; Kansra, Alisha S. ; Pauly, Nicholas P. ; Bristol, Jillian A. ; Hayes, Mitchell ; Ohashi, Makoto ; Casco, Alejandro ; Lee, Denis ; Fogarty, Stuart A. ; Lambert, Paul F. ; Johannsen, Eric C. ; Kenney, Shannon C.
Publisher:United States: Public Library of Science
Journal title:PLoS pathogens, 2022-10, Vol.18 (10), p.e1010868
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Author Correction: The EphA2 receptor is activated through induction of distinct, ligand-dependent oligomeric structures
Author / Creator:Singh, Deo R. ; Kanvinde, Pranjali ; King, Christopher ; Pasquale, Elena B. ; Hristova, Kalina
Publisher:London: Nature Publishing Group UK
Journal title:Communications biology, 2018-04, Vol.1 (1), p.1-1, Article 40
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Inhibition of the Integrated stress response by Epstein-Barr virus oncoprotein LMP1 attenuates epithelial cell differentiation and lytic viral reactivation
Author / Creator:Singh, Deo R ; Zhang, Yitao ; White, Sophia J ; Nelson, Scott E ; Fogarty, Stuart A ; Pawelski, Abigail S ; Kansra, Alisha S ; Kenney, Shannon C
Publisher:United States: Public Library of Science
Journal title:PLoS pathogens, 2025-02, Vol.21 (2), p.e1012934
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ArticlesPeer ReviewedFull Text
Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes
Author / Creator:Deo R Singh ; Scott E Nelson ; Abigail S Pawelski ; Juan A Cantres-Velez ; Alisha S Kansra ; Nicholas P Pauly ; Jillian A Bristol ; Mitchell Hayes ; Makoto Ohashi ; Alejandro Casco ; Denis Lee ; Stuart A Fogarty ; Paul F Lambert ; Eric C Johannsen ; Shannon C Kenney
Publisher:Public Library of Science (PLoS)
Journal title:PLoS pathogens, 2022-10, Vol.18 (10), p.e1010868
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ArticlesPeer ReviewedFull Text
Erratum: Author Correction: The EphA2 receptor is activated through induction of distinct, ligand-dependent oligomeric structures
Author / Creator:Singh, Deo R ; Kanvinde, Pranjali ; King, Christopher ; Pasquale, Elena B ; Hristova, Kalina
Publisher:England
Journal title:Communications biology, 2018, Vol.1, p.40-40
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Latent Epstein-Barr virus infection collaborates with Myc over-expression in normal human B cells to induce Burkitt-like Lymphomas in mice

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The EphA2 receptor is activated through induction of distinct, ligand-dependent oligomeric structures

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Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes

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Author Correction: The EphA2 receptor is activated through induction of distinct, ligand-dependent oligomeric structures

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Inhibition of the Integrated stress response by Epstein-Barr virus oncoprotein LMP1 attenuates epithelial cell differentiation and lytic viral reactivation

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Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes

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Erratum: Author Correction: The EphA2 receptor is activated through induction of distinct, ligand-dependent oligomeric structures

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