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Canonical versus non-canonical transsynaptic signaling of neuroligin 3 tunes development of socialit...

Canonical versus non-canonical transsynaptic signaling of neuroligin 3 tunes development of socialit...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_088ec8c07114426fb988df9303a95e47

Canonical versus non-canonical transsynaptic signaling of neuroligin 3 tunes development of sociality in mice

About this item

Full title

Canonical versus non-canonical transsynaptic signaling of neuroligin 3 tunes development of sociality in mice

Publisher

London: Nature Publishing Group UK

Journal title

Nature communications, 2021-03, Vol.12 (1), p.1848-15, Article 1848

Language

English

Formats

Publication information

Publisher

London: Nature Publishing Group UK

More information

Scope and Contents

Contents

Neuroligin 3 (NLGN3) and neurexins (NRXNs) constitute a canonical transsynaptic cell-adhesion pair, which has been implicated in autism. In autism spectrum disorder (ASD) development of sociality can be impaired. However, the molecular mechanism underlying NLGN3-mediated social development is unclear. Here, we identify non-canonical interactions be...

Alternative Titles

Full title

Canonical versus non-canonical transsynaptic signaling of neuroligin 3 tunes development of sociality in mice

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_doaj_primary_oai_doaj_org_article_088ec8c07114426fb988df9303a95e47

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_088ec8c07114426fb988df9303a95e47

Other Identifiers

ISSN

2041-1723

E-ISSN

2041-1723

DOI

10.1038/s41467-021-22059-6

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