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Distinct macrophage phenotypes skewed by local granulocyte macrophage colony‐stimulating factor (GM‐...

Distinct macrophage phenotypes skewed by local granulocyte macrophage colony‐stimulating factor (GM‐...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_65049c98a25546afab7ccb4124a70405

Distinct macrophage phenotypes skewed by local granulocyte macrophage colony‐stimulating factor (GM‐CSF) and macrophage colony‐stimulating factor (M‐CSF) are associated with tissue destruction and intimal hyperplasia in giant cell arteritis

About this item

Full title

Distinct macrophage phenotypes skewed by local granulocyte macrophage colony‐stimulating factor (GM‐CSF) and macrophage colony‐stimulating factor (M‐CSF) are associated with tissue destruction and intimal hyperplasia in giant cell arteritis

Publisher

Australia: John Wiley & Sons, Inc

Journal title

Clinical & translational immunology, 2020, Vol.9 (9), p.e1164-n/a

Language

English

Formats

Publication information

Publisher

Australia: John Wiley & Sons, Inc

More information

Scope and Contents

Contents

Objective
To determine the presence and spatial distribution of different macrophage phenotypes, governed by granulocyte macrophage colony‐stimulating factor (GM‐CSF) and macrophage colony‐stimulating factor (M‐CSF) skewing signals, in giant cell arteritis (GCA) lesions.
Methods
Temporal artery biopsies (TABs, n = 11) from treatment‐naive...

Alternative Titles

Full title

Distinct macrophage phenotypes skewed by local granulocyte macrophage colony‐stimulating factor (GM‐CSF) and macrophage colony‐stimulating factor (M‐CSF) are associated with tissue destruction and intimal hyperplasia in giant cell arteritis

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_doaj_primary_oai_doaj_org_article_65049c98a25546afab7ccb4124a70405

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_65049c98a25546afab7ccb4124a70405

Other Identifiers

ISSN

2050-0068

E-ISSN

2050-0068

DOI

10.1002/cti2.1164

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