Mic19 depletion impairs endoplasmic reticulum-mitochondrial contacts and mitochondrial lipid metabol...
Mic19 depletion impairs endoplasmic reticulum-mitochondrial contacts and mitochondrial lipid metabolism and triggers liver disease
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Author / Creator
Dong, Jun , Chen, Li , Ye, Fei , Tang, Junhui , Liu, Bing , Lin, Jiacheng , Zhou, Pang-Hu , Lu, Bin , Wu, Min , Lu, Jia-Hong , He, Jing-Jing , Engelender, Simone , Meng, Qingtao , Song, Zhiyin and He, He
Publisher
London: Nature Publishing Group UK
Journal title
Language
English
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Publication information
Publisher
London: Nature Publishing Group UK
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Scope and Contents
Contents
Endoplasmic reticulum (ER)-mitochondria contacts are critical for the regulation of lipid transport, synthesis, and metabolism. However, the molecular mechanism and physiological function of endoplasmic reticulum-mitochondrial contacts remain unclear. Here, we show that Mic19, a key subunit of MICOS (mitochondrial contact site and cristae organizin...
Alternative Titles
Full title
Mic19 depletion impairs endoplasmic reticulum-mitochondrial contacts and mitochondrial lipid metabolism and triggers liver disease
Authors, Artists and Contributors
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Record Identifier
TN_cdi_doaj_primary_oai_doaj_org_article_9ce0791913fe4ff9aa6525cb89bbb6a4
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_9ce0791913fe4ff9aa6525cb89bbb6a4
Other Identifiers
ISSN
2041-1723
E-ISSN
2041-1723
DOI
10.1038/s41467-023-44057-6
