PLAC8-Mediated Activation of NOX4 Signalling Restores Angiogenic Function of Endothelial Colony-Form...
PLAC8-Mediated Activation of NOX4 Signalling Restores Angiogenic Function of Endothelial Colony-Forming Cells in Experimental Hypoxia
About this item
Full title
Author / Creator
Pun, Shun Hay , O’Neill, Karla M. , Edgar, Kevin S. , Gill, Eleanor K. , Moez, Arya , Naderi-Meshkin, Hojjat , Malla, Sudhir B. , Hookham, Michelle B. , Alsaggaf, Mohammed , Madishetti, Vinuthna Vani , Botezatu, Bianca , King, William , Brunssen, Coy , Morawietz, Henning , Dunne, Philip D. , Brazil, Derek P. , Medina, Reinhold J. , Watson, Chris J. and Grieve, David J.
Publisher
Switzerland: MDPI AG
Journal title
Language
English
Formats
Publication information
Publisher
Switzerland: MDPI AG
Subjects
More information
Scope and Contents
Contents
Ischaemic cardiovascular disease is associated with tissue hypoxia as a significant determinant of angiogenic dysfunction and adverse remodelling. While cord blood-derived endothelial colony-forming cells (CB-ECFCs) hold clear therapeutic potential due to their enhanced angiogenic and proliferative capacity, their impaired functionality within the...
Alternative Titles
Full title
PLAC8-Mediated Activation of NOX4 Signalling Restores Angiogenic Function of Endothelial Colony-Forming Cells in Experimental Hypoxia
Authors, Artists and Contributors
Author / Creator
O’Neill, Karla M.
Edgar, Kevin S.
Gill, Eleanor K.
Moez, Arya
Naderi-Meshkin, Hojjat
Malla, Sudhir B.
Hookham, Michelle B.
Alsaggaf, Mohammed
Madishetti, Vinuthna Vani
Botezatu, Bianca
King, William
Brunssen, Coy
Morawietz, Henning
Dunne, Philip D.
Brazil, Derek P.
Medina, Reinhold J.
Watson, Chris J.
Grieve, David J.
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_doaj_primary_oai_doaj_org_article_c401b027209144cda01f2bdf818f87be
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_c401b027209144cda01f2bdf818f87be
Other Identifiers
ISSN
2073-4409
E-ISSN
2073-4409
DOI
10.3390/cells12182220
