METTL3-mediated m6A methylation regulates ovarian cancer progression by recruiting myeloid-derived s...
METTL3-mediated m6A methylation regulates ovarian cancer progression by recruiting myeloid-derived suppressor cells
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Publisher
London: BioMed Central Ltd
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Language
English
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London: BioMed Central Ltd
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Contents
Ovarian cancer (OC) typically develops an immunosuppressive microenvironment by funtional changes of host immune cells. Dysregulated m6A level is associated with cancer progression via the intrinsic oncogenic pathways. However, the role of m6A in regulating host immune cell function during anti-tumor immunity needs comprehensive analysis. This stud...
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METTL3-mediated m6A methylation regulates ovarian cancer progression by recruiting myeloid-derived suppressor cells
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TN_cdi_doaj_primary_oai_doaj_org_article_cd779173a1154f52bd0def7cbe3f43bc
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_cd779173a1154f52bd0def7cbe3f43bc
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ISSN
2045-3701
E-ISSN
2045-3701
DOI
10.1186/s13578-023-01149-6