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METTL3-mediated m6A methylation regulates ovarian cancer progression by recruiting myeloid-derived s...

METTL3-mediated m6A methylation regulates ovarian cancer progression by recruiting myeloid-derived s...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_cd779173a1154f52bd0def7cbe3f43bc

METTL3-mediated m6A methylation regulates ovarian cancer progression by recruiting myeloid-derived suppressor cells

About this item

Full title

METTL3-mediated m6A methylation regulates ovarian cancer progression by recruiting myeloid-derived suppressor cells

Publisher

London: BioMed Central Ltd

Journal title

Cell & bioscience, 2023-11, Vol.13 (1), p.1-202, Article 202

Language

English

Formats

Publication information

Publisher

London: BioMed Central Ltd

More information

Scope and Contents

Contents

Ovarian cancer (OC) typically develops an immunosuppressive microenvironment by funtional changes of host immune cells. Dysregulated m6A level is associated with cancer progression via the intrinsic oncogenic pathways. However, the role of m6A in regulating host immune cell function during anti-tumor immunity needs comprehensive analysis. This stud...

Alternative Titles

Full title

METTL3-mediated m6A methylation regulates ovarian cancer progression by recruiting myeloid-derived suppressor cells

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_doaj_primary_oai_doaj_org_article_cd779173a1154f52bd0def7cbe3f43bc

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_cd779173a1154f52bd0def7cbe3f43bc

Other Identifiers

ISSN

2045-3701

E-ISSN

2045-3701

DOI

10.1186/s13578-023-01149-6

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