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Lack of APLP1 leads to subtle alterations in neuronal morphology but does not affect learning and me...

Lack of APLP1 leads to subtle alterations in neuronal morphology but does not affect learning and me...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_ffbf24cb46bb4306b854fd2685dece4f

Lack of APLP1 leads to subtle alterations in neuronal morphology but does not affect learning and memory

About this item

Full title

Lack of APLP1 leads to subtle alterations in neuronal morphology but does not affect learning and memory

Publisher

Frontiers Media S.A

Journal title

Frontiers in molecular neuroscience, 2022-10, Vol.15, p.1028836-1028836

Language

English

Formats

Publication information

Publisher

Frontiers Media S.A

More information

Scope and Contents

Contents

The amyloid precursor protein APP plays a crucial role in Alzheimer pathogenesis. Its physiological functions, however, are only beginning to be unraveled. APP belongs to a small gene family, including besides APP the closely related amyloid precursor-like proteins APLP1 and APLP2, that all constitute synaptic adhesion proteins. While APP and APLP2...

Alternative Titles

Full title

Lack of APLP1 leads to subtle alterations in neuronal morphology but does not affect learning and memory

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_doaj_primary_oai_doaj_org_article_ffbf24cb46bb4306b854fd2685dece4f

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_doaj_primary_oai_doaj_org_article_ffbf24cb46bb4306b854fd2685dece4f

Other Identifiers

ISSN

1662-5099

E-ISSN

1662-5099

DOI

10.3389/fnmol.2022.1028836

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