Opposite microglial activation stages upon loss of PGRN or TREM 2 result in reduced cerebral glucose...
Opposite microglial activation stages upon loss of PGRN or TREM 2 result in reduced cerebral glucose metabolism
About this item
Full title
Author / Creator
Götzl, Julia K , Brendel, Matthias , Werner, Georg , Parhizkar, Samira , Sebastian Monasor, Laura , Kleinberger, Gernot , Colombo, Alessio‐Vittorio , Deussing, Maximilian , Wagner, Matias , Winkelmann, Juliane , Diehl‐Schmid, Janine , Levin, Johannes , Fellerer, Katrin , Reifschneider, Anika , Bultmann, Sebastian , Bartenstein, Peter , Rominger, Axel , Tahirovic, Sabina , Smith, Scott T , Madore, Charlotte , Butovsky, Oleg , Capell, Anja and Haass, Christian
Publisher
Frankfurt: EMBO Press
Journal title
Language
English
Formats
Publication information
Publisher
Frankfurt: EMBO Press
Subjects
More information
Scope and Contents
Contents
Microglia adopt numerous fates with homeostatic microglia (HM) and a microglial neurodegenerative phenotype (MGnD) representing two opposite ends. A number of variants in genes selectively expressed in microglia are associated with an increased risk for neurodegenerative diseases such as Alzheimer's disease (AD) and frontotemporal lobar degeneratio...
Alternative Titles
Full title
Opposite microglial activation stages upon loss of PGRN or TREM 2 result in reduced cerebral glucose metabolism
Authors, Artists and Contributors
Author / Creator
Brendel, Matthias
Werner, Georg
Parhizkar, Samira
Sebastian Monasor, Laura
Kleinberger, Gernot
Colombo, Alessio‐Vittorio
Deussing, Maximilian
Wagner, Matias
Winkelmann, Juliane
Diehl‐Schmid, Janine
Levin, Johannes
Fellerer, Katrin
Reifschneider, Anika
Bultmann, Sebastian
Bartenstein, Peter
Rominger, Axel
Tahirovic, Sabina
Smith, Scott T
Madore, Charlotte
Butovsky, Oleg
Capell, Anja
Haass, Christian
Identifiers
Primary Identifiers
Record Identifier
TN_cdi_hal_primary_oai_HAL_hal_03704634v1
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_hal_primary_oai_HAL_hal_03704634v1
Other Identifiers
ISSN
1757-4676
E-ISSN
1757-4684
DOI
10.15252/emmm.201809711