Excess soluble CD40L contributes to blood brain barrier permeability in vivo: implications for HIV-a...
Excess soluble CD40L contributes to blood brain barrier permeability in vivo: implications for HIV-associated neurocognitive disorders
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United States: Public Library of Science
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Language
English
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Publisher
United States: Public Library of Science
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Contents
Despite the use of anti-retroviral therapies, a majority of HIV-infected individuals still develop HIV-Associated Neurocognitive Disorders (HAND), indicating that host inflammatory mediators, in addition to viral proteins, may be contributing to these disorders. Consistently, we have previously shown that levels of the inflammatory mediator soluble...
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Full title
Excess soluble CD40L contributes to blood brain barrier permeability in vivo: implications for HIV-associated neurocognitive disorders
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TN_cdi_plos_journals_1327136275
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_plos_journals_1327136275
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ISSN
1932-6203
E-ISSN
1932-6203
DOI
10.1371/journal.pone.0051793
