HSV-1-induced disruption of transcription termination resembles a cellular stress response but selec...
HSV-1-induced disruption of transcription termination resembles a cellular stress response but selectively increases chromatin accessibility downstream of genes
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United States: Public Library of Science
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English
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United States: Public Library of Science
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Lytic herpes simplex virus 1 (HSV-1) infection triggers disruption of transcription termination (DoTT) of most cellular genes, resulting in extensive intergenic transcription. Similarly, cellular stress responses lead to gene-specific transcription downstream of genes (DoG). In this study, we performed a detailed comparison of DoTT/DoG transcriptio...
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HSV-1-induced disruption of transcription termination resembles a cellular stress response but selectively increases chromatin accessibility downstream of genes
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TN_cdi_plos_journals_2025706764
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_plos_journals_2025706764
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1553-7374,1553-7366
E-ISSN
1553-7374
DOI
10.1371/journal.ppat.1006954