Activation of NRF2 by dexamethasone in ataxia telangiectasia cells involves KEAP1 inhibition but not...
Activation of NRF2 by dexamethasone in ataxia telangiectasia cells involves KEAP1 inhibition but not the inhibition of p38
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United States: Public Library of Science
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English
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United States: Public Library of Science
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Oxidative stress has been shown to play a crucial role in the pathophysiology of the neurodegenerative disease Ataxia Telangiectasia. We have recently demonstrated that Dexamethasone treatment is able to counteract the oxidative state by promoting nuclear factor erythroid 2-related factor 2 (NRF2) nuclear accumulation. However, substantial gaps rem...
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Activation of NRF2 by dexamethasone in ataxia telangiectasia cells involves KEAP1 inhibition but not the inhibition of p38
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TN_cdi_plos_journals_2227830742
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_plos_journals_2227830742
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ISSN
1932-6203
E-ISSN
1932-6203
DOI
10.1371/journal.pone.0216668
