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Prenatal Exposure to a Cannabinoid Agonist Produces Memory Deficits Linked to Dysfunction in Hippoca...

Prenatal Exposure to a Cannabinoid Agonist Produces Memory Deficits Linked to Dysfunction in Hippoca...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pnas_primary_100_8_4915

Prenatal Exposure to a Cannabinoid Agonist Produces Memory Deficits Linked to Dysfunction in Hippocampal Long-Term Potentiation and Glutamate Release

About this item

Full title

Prenatal Exposure to a Cannabinoid Agonist Produces Memory Deficits Linked to Dysfunction in Hippocampal Long-Term Potentiation and Glutamate Release

Publisher

United States: National Academy of Sciences

Journal title

Proceedings of the National Academy of Sciences - PNAS, 2003-04, Vol.100 (8), p.4915-4920

Language

English

Formats

Publication information

Publisher

United States: National Academy of Sciences

More information

Scope and Contents

Contents

To investigate the possible long-term consequences of gestational exposure to cannabinoids on cognitive functions, pregnant rats were administered with the CB1 receptor agonist WIN 55,212-2 (WIN), at a dose (0.5 mg/kg) that causes neither malformations nor overt signs of toxicity. Prenatal WIN exposure induced a disruption of memory retention in 40...

Alternative Titles

Full title

Prenatal Exposure to a Cannabinoid Agonist Produces Memory Deficits Linked to Dysfunction in Hippocampal Long-Term Potentiation and Glutamate Release

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pnas_primary_100_8_4915

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pnas_primary_100_8_4915

Other Identifiers

ISSN

0027-8424

E-ISSN

1091-6490

DOI

10.1073/pnas.0537849100

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