Prenatal Exposure to a Cannabinoid Agonist Produces Memory Deficits Linked to Dysfunction in Hippoca...
Prenatal Exposure to a Cannabinoid Agonist Produces Memory Deficits Linked to Dysfunction in Hippocampal Long-Term Potentiation and Glutamate Release
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United States: National Academy of Sciences
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Language
English
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United States: National Academy of Sciences
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To investigate the possible long-term consequences of gestational exposure to cannabinoids on cognitive functions, pregnant rats were administered with the CB1 receptor agonist WIN 55,212-2 (WIN), at a dose (0.5 mg/kg) that causes neither malformations nor overt signs of toxicity. Prenatal WIN exposure induced a disruption of memory retention in 40...
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Full title
Prenatal Exposure to a Cannabinoid Agonist Produces Memory Deficits Linked to Dysfunction in Hippocampal Long-Term Potentiation and Glutamate Release
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TN_cdi_pnas_primary_100_8_4915
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pnas_primary_100_8_4915
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ISSN
0027-8424
E-ISSN
1091-6490
DOI
10.1073/pnas.0537849100