Endothelin-1 Inhibits Background Two-Pore Domain Channel TASK-1 in Primary Human Pulmonary Artery Sm...
Endothelin-1 Inhibits Background Two-Pore Domain Channel TASK-1 in Primary Human Pulmonary Artery Smooth Muscle Cells
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Publisher
United States: Am Thoracic Soc
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Language
English
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United States: Am Thoracic Soc
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Contents
Endothelin (ET)-1 causes long-lasting vasoconstriction and vascular remodeling by interacting with specific G-protein-coupled receptors in pulmonary artery smooth muscle cells (PASMCs), and thus plays an important role in the pathophysiology of pulmonary arterial hypertension. The two-pore domain K(+) channel, TASK-1, controls the resting membrane...
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Full title
Endothelin-1 Inhibits Background Two-Pore Domain Channel TASK-1 in Primary Human Pulmonary Artery Smooth Muscle Cells
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TN_cdi_proquest_journals_207655603
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_207655603
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ISSN
1044-1549
E-ISSN
1535-4989
DOI
10.1165/rcmb.2008-0412OC
