Kinetic fingerprints differentiate anti-Aβ therapies
Kinetic fingerprints differentiate anti-Aβ therapies
About this item
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Author / Creator
Linse, Sara , Scheidt, Tom , Bernfur, Katja , Vendruscolo, Michele , Dobson, Christopher M , Cohen, Samuel Ia , Sileikis, Eimantas , Lundquist, Martin , Fang Qian , O'malley, Tiernan , Bussiere, Thierry , Weinreb, Paul H , Xu, Catherine K , Meisl, Georg , Devenish, Sean , Knowles, Tuomas Pj and Hansson, Oskar
Publisher
Cold Spring Harbor: Cold Spring Harbor Laboratory Press
Journal title
Language
English
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Publisher
Cold Spring Harbor: Cold Spring Harbor Laboratory Press
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More information
Scope and Contents
Contents
Alzheimer's disease affects nearly 50 million people worldwide with an overall cost corresponding to more than 1% of the global economy. The amyloid cascade hypothesis, according to which the misfolding and self-assembly of the amyloid-β peptide (Aβ) into oligomeric and fibrillar aggregates is a causative pathogenic process in this disease (1,2), h...
Alternative Titles
Full title
Kinetic fingerprints differentiate anti-Aβ therapies
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TN_cdi_proquest_journals_2366704577
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_journals_2366704577
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E-ISSN
2692-8205
DOI
10.1101/815308
How to access this item
https://www.proquest.com/docview/2366704577?pq-origsite=primo&accountid=13902
