Nitric oxide controls the immunopathology of tuberculosis by inhibiting NLRP3 inflammasome–dependent...
Nitric oxide controls the immunopathology of tuberculosis by inhibiting NLRP3 inflammasome–dependent processing of IL-1β
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New York: Nature Publishing Group US
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Language
English
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New York: Nature Publishing Group US
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Contents
Chronic infections can result in harmful production of the proinflammatory cytokine IL-1 generated via the NLRP3 inflammasome. Sassetti
et al
. demonstrate that IL-1 activated by
Mycobacteria
results in nitrosylation and consequent regulation of the NLRP3 inflammasome.
Interleukin 1 (IL-1) is an important mediator of innate immunity...
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Full title
Nitric oxide controls the immunopathology of tuberculosis by inhibiting NLRP3 inflammasome–dependent processing of IL-1β
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TN_cdi_proquest_miscellaneous_1291605695
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_1291605695
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ISSN
1529-2908
E-ISSN
1529-2916
DOI
10.1038/ni.2474
