Epigallocatechin Gallate Attenuates β-Amyloid Generation and Oxidative Stress Involvement of PPARγ i...
Epigallocatechin Gallate Attenuates β-Amyloid Generation and Oxidative Stress Involvement of PPARγ in N2a/APP695 Cells
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New York: Springer US
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English
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New York: Springer US
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The accumulation of β-amyloid (Aβ) peptide plaques is a major pathogenic event in Alzheimer’s disease (AD). Aβ is a cleaved fragment of APP via BACE1, which is the rate-limiting enzyme in APP processing and Aβ generation. Nuclear receptor peroxisome proliferator-activated receptor gamma (PPARγ) is considered to be a potential target for AD treatmen...
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Epigallocatechin Gallate Attenuates β-Amyloid Generation and Oxidative Stress Involvement of PPARγ in N2a/APP695 Cells
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TN_cdi_proquest_miscellaneous_1877849474
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_1877849474
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ISSN
0364-3190
E-ISSN
1573-6903
DOI
10.1007/s11064-016-2093-8