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The role of labile Zn2+ and Zn2+–transporters in the pathophysiology of mitochondria dysfunction in...

The role of labile Zn2+ and Zn2+–transporters in the pathophysiology of mitochondria dysfunction in...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_2463598376

The role of labile Zn2+ and Zn2+–transporters in the pathophysiology of mitochondria dysfunction in cardiomyocytes

About this item

Full title

The role of labile Zn2+ and Zn2+–transporters in the pathophysiology of mitochondria dysfunction in cardiomyocytes

Author / Creator

Publisher

New York: Springer US

Journal title

Molecular and cellular biochemistry, 2021-02, Vol.476 (2), p.971-989

Language

English

Formats

Publication information

Publisher

New York: Springer US

More information

Scope and Contents

Contents

An important energy supplier of cardiomyocytes is mitochondria, similar to other mammalian cells. Studies have demonstrated that any defect in the normal processes controlled by mitochondria can lead to abnormal ROS production, thereby high oxidative stress as well as lack of ATP. Taken into consideration, the relationship between mitochondrial dys...

Alternative Titles

Full title

The role of labile Zn2+ and Zn2+–transporters in the pathophysiology of mitochondria dysfunction in cardiomyocytes

Authors, Artists and Contributors

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_proquest_miscellaneous_2463598376

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_2463598376

Other Identifiers

ISSN

0300-8177

E-ISSN

1573-4919

DOI

10.1007/s11010-020-03964-8

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