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Dysfunction of lipid sensor GPR120 leads to obesity in both mouse and human

Dysfunction of lipid sensor GPR120 leads to obesity in both mouse and human

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_928910242

Publication information

Publisher

London: Nature Publishing Group UK

More information

Scope and Contents

Contents

Mice deficient in the lipid sensor GPR120 develop obesity, glucose intolerance and fatty liver when fed a high-fat diet, and a loss-of-function variant in the
GPR120
gene strongly contributes to increased obesity in human.
A signal for obesity
The G-protein-coupled receptor GPR120 is a receptor for free fatty acids, and is involved in h...

Alternative Titles

Full title

Dysfunction of lipid sensor GPR120 leads to obesity in both mouse and human

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_proquest_miscellaneous_928910242

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_928910242

Other Identifiers

ISSN

0028-0836

E-ISSN

1476-4687

DOI

10.1038/nature10798

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