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Bradykinin Metabolism and Drug-Induced Angioedema

Bradykinin Metabolism and Drug-Induced Angioedema

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_10380452

Bradykinin Metabolism and Drug-Induced Angioedema

About this item

Full title

Bradykinin Metabolism and Drug-Induced Angioedema

Publisher

Switzerland: MDPI AG

Journal title

International journal of molecular sciences, 2023-07, Vol.24 (14), p.11649

Language

English

Formats

Publication information

Publisher

Switzerland: MDPI AG

More information

Scope and Contents

Contents

Bradykinin (BK) metabolism and its receptors play a central role in drug-induced angioedema (AE) without urticaria through increased vascular permeability. Many cardiovascular and diabetic drugs may cause BK-mediated AE. Angiotensin-converting enzyme inhibitors (ACEIs) and neprilysin inhibitors impair BK catabolism. Dipeptidyl peptidase-IV (DPP-IV)...

Alternative Titles

Full title

Bradykinin Metabolism and Drug-Induced Angioedema

Authors, Artists and Contributors

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_10380452

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_10380452

Other Identifiers

ISSN

1422-0067,1661-6596

E-ISSN

1422-0067

DOI

10.3390/ijms241411649

How to access this item