Log in to save to my catalogue

β-Arrestin2-Mediated Inotropic Effects of the Angiotensin II Type 1A Receptor in Isolated Cardiac My...

β-Arrestin2-Mediated Inotropic Effects of the Angiotensin II Type 1A Receptor in Isolated Cardiac My...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_1637574

β-Arrestin2-Mediated Inotropic Effects of the Angiotensin II Type 1A Receptor in Isolated Cardiac Myocytes

About this item

Full title

β-Arrestin2-Mediated Inotropic Effects of the Angiotensin II Type 1A Receptor in Isolated Cardiac Myocytes

Publisher

United States: National Academy of Sciences

Journal title

Proceedings of the National Academy of Sciences - PNAS, 2006-10, Vol.103 (44), p.16284-16289

Language

English

Formats

Publication information

Publisher

United States: National Academy of Sciences

More information

Scope and Contents

Contents

The G protein-coupled receptor kinases (GRKs) and β-arrestins, families of molecules essential to the desensitization of G proteindependent signaling via seven-transmembrane receptors (7TMRs), have been recently shown to also transduce G protein-independent signals from receptors. However, the physiologic consequences of this G protein-independent,...

Alternative Titles

Full title

β-Arrestin2-Mediated Inotropic Effects of the Angiotensin II Type 1A Receptor in Isolated Cardiac Myocytes

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_1637574

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_1637574

Other Identifiers

ISSN

0027-8424

E-ISSN

1091-6490

DOI

10.1073/pnas.0607583103

How to access this item