Reversal of learning deficits in a Tsc2+/− mouse model of tuberous sclerosis
Reversal of learning deficits in a Tsc2+/− mouse model of tuberous sclerosis
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New York: Nature Publishing Group US
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Language
English
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New York: Nature Publishing Group US
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Contents
Tuberous sclerosis is a neurological disorder associated with seizures and cognitive dysfunction. Alcino Silva and his colleagues find that rapamycin, an inhibitor of the mTOR signaling pathway, can ameliorate cognitive deficits in a mouse model of the disease.
Tuberous sclerosis is a single-gene disorder caused by heterozygous mutations in the<...
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Reversal of learning deficits in a Tsc2+/− mouse model of tuberous sclerosis
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TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_2664098
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https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_2664098
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ISSN
1078-8956
E-ISSN
1546-170X
DOI
10.1038/nm1788
