Log in to save to my catalogue

Increased Expression of TrkB and Capzb2 Accompanies Preserved Cognitive Status in Early Alzheimer Di...

Increased Expression of TrkB and Capzb2 Accompanies Preserved Cognitive Status in Early Alzheimer Di...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3398703

Increased Expression of TrkB and Capzb2 Accompanies Preserved Cognitive Status in Early Alzheimer Disease Pathology

About this item

Full title

Increased Expression of TrkB and Capzb2 Accompanies Preserved Cognitive Status in Early Alzheimer Disease Pathology

Publisher

Hagerstown, MD: American Association of Neuropathologists, Inc

Journal title

Journal of neuropathology and experimental neurology, 2012-07, Vol.71 (7), p.654-664

Language

English

Formats

Publication information

Publisher

Hagerstown, MD: American Association of Neuropathologists, Inc

More information

Scope and Contents

Contents

ABSTRACTBrain-derived neurotrophic factor (BDNF) and its receptor tyrosine kinase B (TrkB) may influence brain reserve, the ability of the brain to tolerate pathological changes without significant decline in function. Here, we explore whether a specifically vulnerable population of human neurons shows a compensatory response to the neuropathologic...

Alternative Titles

Full title

Increased Expression of TrkB and Capzb2 Accompanies Preserved Cognitive Status in Early Alzheimer Disease Pathology

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3398703

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3398703

Other Identifiers

ISSN

0022-3069

E-ISSN

1554-6578,0022-3069

DOI

10.1097/NEN.0b013e31825d06b7

How to access this item