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Regulation of Transforming Growth Factor-β1-driven Lung Fibrosis by Galectin-3

Regulation of Transforming Growth Factor-β1-driven Lung Fibrosis by Galectin-3

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3410728

Regulation of Transforming Growth Factor-β1-driven Lung Fibrosis by Galectin-3

About this item

Full title

Regulation of Transforming Growth Factor-β1-driven Lung Fibrosis by Galectin-3

Publisher

New York, NY: American Thoracic Society

Journal title

American journal of respiratory and critical care medicine, 2012-03, Vol.185 (5), p.537-546

Language

English

Formats

Publication information

Publisher

New York, NY: American Thoracic Society

More information

Scope and Contents

Contents

Idiopathic pulmonary fibrosis (IPF) is a chronic dysregulated response to alveolar epithelial injury with differentiation of epithelial cells and fibroblasts into matrix-secreting myofibroblasts resulting in lung scaring. The prognosis is poor and there are no effective therapies or reliable biomarkers. Galectin-3 is a β-galactoside binding lectin...

Alternative Titles

Full title

Regulation of Transforming Growth Factor-β1-driven Lung Fibrosis by Galectin-3

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3410728

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_3410728

Other Identifiers

ISSN

1073-449X

E-ISSN

1535-4970

DOI

10.1164/rccm.201106-0965oc

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