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Abnormal Population Responses in the Somatosensory Cortex of Alzheimer’s Disease Model Mice

Abnormal Population Responses in the Somatosensory Cortex of Alzheimer’s Disease Model Mice

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4832196

Abnormal Population Responses in the Somatosensory Cortex of Alzheimer’s Disease Model Mice

About this item

Full title

Abnormal Population Responses in the Somatosensory Cortex of Alzheimer’s Disease Model Mice

Publisher

London: Nature Publishing Group UK

Journal title

Scientific reports, 2016-04, Vol.6 (1), p.24560-24560, Article 24560

Language

English

Formats

Publication information

Publisher

London: Nature Publishing Group UK

More information

Scope and Contents

Contents

Alzheimer’s disease (AD) is the most common form of dementia. One of the neuropathological hallmarks of AD is the accumulation of amyloid-β plaques. Overexpression of human amyloid precursor protein in transgenic mice induces hippocampal and neocortical amyloid-β accumulation and plaque deposition that increases with age. The impact of these effect...

Alternative Titles

Full title

Abnormal Population Responses in the Somatosensory Cortex of Alzheimer’s Disease Model Mice

Authors, Artists and Contributors

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4832196

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4832196

Other Identifiers

ISSN

2045-2322

E-ISSN

2045-2322

DOI

10.1038/srep24560

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