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Glial contributions to visceral pain: implications for disease etiology and the female predominance...

Glial contributions to visceral pain: implications for disease etiology and the female predominance...

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5048206

Glial contributions to visceral pain: implications for disease etiology and the female predominance of persistent pain

About this item

Full title

Glial contributions to visceral pain: implications for disease etiology and the female predominance of persistent pain

Publisher

London: Nature Publishing Group UK

Journal title

Translational psychiatry, 2016-09, Vol.6 (9), p.e888-e888

Language

English

Formats

Publication information

Publisher

London: Nature Publishing Group UK

More information

Scope and Contents

Contents

In the central nervous system, bidirectional signaling between glial cells and neurons (‘neuroimmune communication’) facilitates the development of persistent pain. Spinal glia can contribute to heightened pain states by a prolonged release of neurokine signals that sensitize adjacent centrally projecting neurons. Although many persistent pain cond...

Alternative Titles

Full title

Glial contributions to visceral pain: implications for disease etiology and the female predominance of persistent pain

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5048206

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_5048206

Other Identifiers

ISSN

2158-3188

E-ISSN

2158-3188

DOI

10.1038/tp.2016.168

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