FTLD Patient–Derived Fibroblasts Show Defective Mitochondrial Function and Accumulation of p62
FTLD Patient–Derived Fibroblasts Show Defective Mitochondrial Function and Accumulation of p62
About this item
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Publisher
New York: Springer US
Journal title
Language
English
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Publisher
New York: Springer US
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Scope and Contents
Contents
Frontotemporal lobar degeneration (FTLD) is a clinically, genetically, and neuropathologically heterogeneous group of neurodegenerative syndromes, leading to progressive cognitive dysfunction and frontal and temporal atrophy.
C9orf72
hexanucleotide repeat expansion (C9-HRE) is the most common genetic cause of FTLD, but pathogenic mechanisms u...
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Full title
FTLD Patient–Derived Fibroblasts Show Defective Mitochondrial Function and Accumulation of p62
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TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_8599259
Permalink
https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_8599259
Other Identifiers
ISSN
0893-7648
E-ISSN
1559-1182
DOI
10.1007/s12035-021-02475-x
