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Catalpol Ameliorates Oxidative Stress and Neuroinflammation after Traumatic Brain Injury in Rats

Catalpol Ameliorates Oxidative Stress and Neuroinflammation after Traumatic Brain Injury in Rats

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_2730643553

Catalpol Ameliorates Oxidative Stress and Neuroinflammation after Traumatic Brain Injury in Rats

About this item

Full title

Catalpol Ameliorates Oxidative Stress and Neuroinflammation after Traumatic Brain Injury in Rats

Publisher

New York: Springer US

Journal title

Neurochemical research, 2023-02, Vol.48 (2), p.681-695

Language

English

Formats

Publication information

Publisher

New York: Springer US

More information

Scope and Contents

Contents

Oxidative stress and neuroinflammation are deemed the prime causes of neurological damage after traumatic brain injury (TBI). Catalpol, an active ingredient of
Rehmannia glutinosa
, has been suggested to possess antioxidant and anti-inflammatory properties. This study was designed to investigate the protective effects of catalpol against TBI...

Alternative Titles

Full title

Catalpol Ameliorates Oxidative Stress and Neuroinflammation after Traumatic Brain Injury in Rats

Authors, Artists and Contributors

Identifiers

Primary Identifiers

Record Identifier

TN_cdi_proquest_miscellaneous_2730643553

Permalink

https://devfeature-collection.sl.nsw.gov.au/record/TN_cdi_proquest_miscellaneous_2730643553

Other Identifiers

ISSN

0364-3190

E-ISSN

1573-6903

DOI

10.1007/s11064-022-03796-6

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